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Best Brief – Rex E. Lee Moot Court Competition
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Professional Profile
Peter Hecker practices intellectual property law with an emphasis on patent prosecution in the biotechnology industry. He received his J.D. from Brigham Young University - J. Reuben Clark Law School where he served as an executive editor for the BYU Law Review and participated in the ABA’s national moot court competition. Prior to his legal education, Peter received a B.S. in Molecular Biology from Brigham Young University, and then a Ph.D. in Molecular Medicine from University of Maryland, Baltimore.
Additionally, he worked as a postdoctoral researcher for 1 year at Washington University in St. Louis, and then for another year at University of California, San Francisco. His graduate and postdoctoral research was focused on cardiovascular metabolism. Peter worked as a summer associate at the firm in 2016 and joined the firm in 2017.
Specializing In
Cases, Articles, Speeches & Seminars
Law Pubications
Peter A. Hecker, Reasonable Limits on Antitrust Liability for Reverse Payment Patent Settlements, 11 J. Health & Life Sciences L. 108 (2017).
Peter Hecker, How an Old Non-Statutory Doctrine Got Worked into the § 101 Test for Patent Eligibility, 99 J. Pat. & Trademark Off. Soc’y 4 (2017).
Peter A. Hecker, Comment, Postdoc Payback: A Call for Reform, 31 BYU J. Pub. L. 269 (2016).
Peter A. Hecker, The NRSA Payback Requirement, 4 PostDoc J. 41 (2016).
Science Publications
Ackermann MA, King B, Lieberman NAP, Bobbili PJ, Rudloff M, Berndsen CE, Wright NT, Hecker PA, Kontrogianni-Konstantopoulos A. Novel obscurins mediate cardiomyocyte adhesion and size via the PI3K/AKT/mTOR signaling pathway. Journal of Molecular and Cellular Cardiology. 2017 Oct;111:27-39.
Hu LR, Ackermann MA, Hecker PA, Prosser BL, King B, O'Connell KA, Grogan A, Meyer LC, Berndsen CE, Wright NT, Jonathan Lederer W, Kontrogianni-Konstantopoulos A. Deregulated Ca2+ cycling underlies the development of arrhythmia and heart disease due to mutant obscurin. Science Advances. 2017 Jun 7;3(6):e1603081.
Matkovich SJ, Dorn GW 2nd, Grossenheider TC, Hecker PA. Cardiac disease status dictates functional mRNA targeting profiles of individual microRNAs. Circulation: Cardiovascular Genetics. 2015 Dec;8(6):774-84.
Shekar KC, Li L, Dabkowski ER, Xu W, Ribeiro RF Jr, Hecker PA, Recchia FA, Sadygov RG, Willard B, Kasumov T, Stanley WC. Cardiac mitochondrial proteome dynamics with heavy water reveals stable rate of mitochondrial protein synthesis in heart failure despite decline in mitochondrial oxidative capacity. Journal of Molecular and Cellular Cardiology. 2014 Oct;75:88-97.
Ribeiro RF Jr, Dabkowski ER, O'Connell KA, Xu W, Galvao Tde F, Hecker PA, Shekar KC, Stefanon I, Stanley WC. Effect of a high-protein diet on development of heart failure in response to pressure overload. Applied Physiology, Nutrition, and Metabolism. 2014 Feb;39(2):238-47.
Dabkowski ER, O’Connell KA, Xu W, Ribeiro RF, Hecker PA, Shekar KC, Daneault C, Des Rosiers C, Stanley WC. Docosahexaenoic acid supplementation alters key properties of cardiac mitochondria and modestly attenuates development of left ventricular dysfunction in pressure overload-induced heart failure. Cardiovascular Drugs and Therapy. 2013 Dec;27(6):499-510.
Wang H, Sreenivasan U, Gong DW, O'Connell KA, Dabkowski ER, Hecker PA, Ionica N, Konig M, Mahurkar A, Sun Y, Stanley WC, Sztalryd C. Cardiomyocyte specific perilipin 5 over expression leads to myocardial steatosis, and modest cardiac dysfunction. Journal of Lipid Research. 2013 Apr;54(4):953-65. An editorial highlighted the findings of this work.
Hecker PA, Leopold JA, Gupte SA, Recchia FA, Stanley WC. Impact of glucose 6-phosphate dehydrogenase deficiency on the pathophysiology of cardiovascular disease. The American Journal of Physiology - Heart and Circulatory Physiology. Review. 2013 Feb;304(4):H491-500. I was interviewed as part of a podcast about this article, and it was highlighted on the journal's website.
Asemu G, O'Connell KA, Cox JW, Dabkowski ER, Xu W, Ribeiro RF, Shekar KC, Hecker PA, Rastogi S, Sabbah HN, Hoppel CL, Stanley WC. Enhanced resistance to permeability transition in interfibrillar cardiac mitochondria in dogs: effects of aging and long-term aldosterone infusion. The American Journal of Physiology - Heart and Circulatory Physiology. 2013 Feb;304(4):H514-28.
Hecker PA, Lionetti V, Ribeiro RF Jr, Rastogi S, Brown BH, O’Connell KA, Cox JW, Shekar KC, Gamble D, Sabbah HN, Leopold JA, Gupte SA, Recchia FA, Stanley WC. Glucose-6-phosphate dehydrogenase deficiency increases redox stress and moderately accelerates the development of heart failure. Circulation: Heart Failure. 2013 Jan 1;6(1):118-26.
Hu Y, Matkovich SJ, Hecker PA, Zhang Y, Edwards J, Dorn GW 2nd. Epitranscriptional orchestration of genetic reprogramming is an emergent property of stress-regulated cardiac microRNAs. Proceedings of the National Academy of Sciences. 2012 Nov 27;109(48):19864-9.
Galvao TF, Khairallah RJ, Dabkowski ER, Brown BH, Hecker PA, O’Connell KA, O’Shea KM, Sabbah HN, Rastogi S, Daneault C, Des Rosiers C, Stanley WC. Marine n3 polyunsaturated fatty acids enhance resistance to mitochondrial permeability transition in heart failure, but do not improve survival. The American Journal of Physiology - Heart and Circulatory Physiology. 2013 Jan;304(1):H12-21.
Rawat DK, Hecker P, Watanabe M, Levy RJ, Okada T, Edwards JG, Gupte SA. Glucose-6-Phosphate Dehydrogenase and NADPH redox regulates cardiac myocyte L-type calcium channel activity and myocardial contractile function. PLOS ONE. 2012;7(10):e45365.
Hecker PA, Mapanga RF, Kimar CP, Ribeiro RF Jr, Brown BH, O’Connell KA, Cox JW, Shekar KC, Asemu G, Essop MF, Stanley WC. Effects of glucose 6-phosphate dehydrogenase deficiency on the metabolic and cardiac responses to obesogenic or high fructose diets. American Journal of Physiology - Endocrinology and Metabolism. 2012 Oct;303(8):E959-72. This manuscript was featured on the journal’s website as an Editor’s Pick.
Hecker PA, Galavao TF, O’Shea KM, Brown BH, Henderson R, Riggle H, Gupte SA, Stanley WC. High sugar intake does not adversely affect metabolic and cardiac dysfunction in genetic cardiomyopathy. Nutrition. 2012 May;28(5):520-6.
Galvao TF, Brown BH, Hecker PA, O’Connell K, O’Shea KM, Sabbah HN, Rastogi S, Daneault C, Des Rosiers C, Stanley WC. High intake of saturated fat, but not polyunsaturated fat, improves survival in heart failure despite persistent mitochondrial defects. Cardiovascular Research. 2012 Jan 1;93(1):24-32. An editorial highlighted the findings of this work.
Hecker PA, O’Shea KM, Galvao TF, Brown BH, Stanley WC. Role of adiponectin in the development of high-fat diet-induced metabolic abnormalities in mice. Hormone and Metabolic Research. 2011 Feb;43(2):100-5.
O'Shea KM, Chess DJ, Khairallah RJ, Hecker PA, Lei B, Walsh K, Des Rosiers C, Stanley WC. Omega-3 polyunsaturated fatty acids prevent pressure overload-induced ventricular dilation and decrease in mitochondrial enzymes despite no change in adiponectin. Lipids in Health and Disease. 2010 Sep 6;9:95.
Khairallah RJ, Sparagna GC, Khanna N, O'Shea KM, Hecker PA, Kristian T, Fiskum G, Des Rosiers C, Polster BM, Stanley WC. Dietary supplementation with docosahexaenoic acid, but not eicosapentaenoic acid, dramatically alters cardiac mitochondrial phospholipid fatty acid composition and prevents permeability transition. Biochimica et Biophysica Acta - Bioenergetics. 2010 Aug;1797(8):1555-62.
O'Shea KM, Chess DJ, Khairallah RJ, Rastogi S, Hecker PA, Sabbah HN, Walsh K, Stanley WC. Effects of adiponectin deficiency on structural and metabolic remodeling in mice subjected to pressure overload. The American Journal of Physiology - Heart and Circulatory Physiology. 2010 Jun;298(6):H1639-45.
O'Shea KM, Khairallah RJ, Sparagna GC, Xu W, Hecker PA, Robillard-Frayne I, Des Rosiers C, Kristian T, Murphy RC, Fiskum G, Stanley WC. Dietary omega-3 fatty acids alter cardiac mitochondrial phospholipid composition and delay Ca2+-induced permeability transition. Journal of Molecular and Cellular Cardiology. 2009 Dec;47(6):819-27.
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